By Paul K. Buxton
(BMJ Books) Royal Infirmary, united kingdom. deals a concise review of the entire necessities of dermatology, written for non-dermatologists and first care physicians. Discusses a number of universal stipulations comparable to psoriasis, eczema, zits and rosacea, bacterial an infection, viral an infection, tropical dermatology, and extra. considerable colour photographs. prior version: c1998. Softcover.
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Extra resources for ABC of Dermatology
Blistering eruptions • Barbiturates • Sulphonamides • Iodines or bromides • Chlorpropamide • Salicylates • Phenylbutazone Lichen planus like reactions • Chloroquine • Chlorothiazide • Chlorpropamide Photosensitivity (seen on areas exposed to light) • Thiazide diuretics • Sulphonamides • Tetracyclines Vasculitis • Inflammation around dilated capillaries and small blood vessels • A common component of the erythemas • May occur as red macules and papules with necrotic lesions on the extremities • In children a purpuric type (Henoch–Schönlein purpura) occurs in association with nephritis • Systemic lesions may occur, with renal, joint, gastrointestinal, and central nervous system involvement Purpura Vasculitis Is seen on the skin as a result of: • Thrombocytopenia—platelet deficiency • Senile purpura—due to shearing of capillaries as a result of defective supporting connective tissue • Purpura in patients on corticosteroid treatment—similar to senile purpura • Schamberg’s disease—brown macules and red spots resembling cayenne pepper on the legs of men • Associated vasculitis Some conditions associated with vasculitis • Infection—streptococcal, hepatitis • Drugs—numerous, including sulphonamides, penicillin, phenothiazine, phenacitin • Chemicals—insecticides, weed killers, phenolic compounds • Connective tissue diseases—systemic lupus erythematosus, rheumatoid arthritis • Lymphoma and leukaemia • Dysproteinaemias Acute vasculitis with necrosis Necrotising angiitis 37 ABC of Dermatology Urticaria In this condition itching red weals develop; they resemble the effects of stinging nettle (Urtica dioica) on the skin.
The lesions tend to develop from the hair follicles. It is a persistent condition that varies in severity. Clinically and pathologically the condition has features of both psoriasis and eczema. There is thickening of the epidermis with some of the inflammatory changes of psoriasis and the intercellular oedema of eczema. Parakeratosis—the presence of nuclei above the basement layer—may be noticeable. Recently, increased numbers of Pityrosporum ovale organisms have been reported. Treatment Topical steroids produce a rapid improvement, but not permanent clearing.
Treatment is simple: selenium sulphide shampoo applied regularly with ample water while showering or bathing will clear the infection. The colour change may take some time to clear. A mica scale pityriasis lichenoides Pityriasis versicolor skin lesions 31 ABC of Dermatology Ketoconazole shampoo is an effective alternative. Oral terbinafine, which is very effective in other fungal infections, has no effect. Desquamating stage of generalised erythema Any extensive acute erythema, from the erythroderma of psoriasis to a penicillin rash, commonly shows a stage of shedding large flakes of skin—desquamation—as it resolves.
ABC of Dermatology by Paul K. Buxton